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H pylori essay conclusion
In approximately 20 of infected individuals, infection with. The geographical distribution of Helicobacter pylori differs amongst developing and developed countries that is 80 in developing countries like India and 20 in developed countries Mitchell HM 1998, Pounder RR 1995, Parsonnet J 1995). It is the main cause of peptic ulcers, and it can also cause gastritis and stomach cancer. The discovery of the role of Helicobacter pylori has entirely changed our views and approach to john donne essays the treatment of patients with stomach disorders.
In 1994, a consensus conference convened by The National Institute of Health concl uded that lori was a major cause of peptic ulcer disease and. The discovery of Helicobacter pylori has revolutionized the pathophysiological and clinical approach to gastric and duodenal ulcer. Since the first paper. A few clicks will bring us Abercrombie s paper where we can re ad the.
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Patients infected with CagA strains usually have a higher inflammatory response and are significantly more at risk of developing a symptomatic outcome (Torres., 2009). Most infections are believed to be acquired during childhood and can persist for decades (Blaser M J 1995). Pylori strains isolated from patients in Colombia, South Korea, and Japan, Lu et al reported that infection with dupA-positive strains is significantly associated with duodenal ulceration but negatively associated with gastric cancer (Lu H et al 2005). The d region is located between the i and m region and is typed as d1 if there is no deletion or as d2 if a 69-89 bp deletion is present (Ogiwara H et al 2009). 2008 Michelle Wiepjes.4.2 Physiology, helicobacter pylori is a microaerophillic organism that is, it thrives best in low oxygen environment (Helicobacter writing academic papers in third person foundation 2006). VacA contains four identified variable regions each of which are subdivided into subtypes. Almost routine diagnostics and treatment of gastritis, gastric ulcer or cancer has been replaced by studies in epidemiology, isolation and eradication of a single bacterium. Analysis of the primary sequence and structural organization of the 3' region of the cagA gene showed that there are four subtypes of the gene namely cagA type A-D (Azuma T 2004). CagA protein is one of the best studied virulence factor of lori, the toxin is 120-145 kDa in size and is encoded by the cagA gene which is the part of a 40 kb DNA segment cag pathogenicity island (cagPAI) (Censini S et al 1996). On this page, basics, learn More, see, Play and Learn. Under stress it loses its spiral morphology and eventually become coccoid are not culturable (Bode et al, 1993, Nilius et al 1993).